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DQJD reduces CCH‐induced neuronal death and inflammation levels in vivo. (A) HE staining of cortex (scale bar = 100 μm) and hippocampal CA1 (scale bar = 100 μm). (B) The number of neurons in the cortex and hippocampal CA1 was evaluated by Image J software ( n = 3). (C) Serum levels of TNF‐α, COX2, and ICAM‐1 were measured by ELISA (mean ± SD, n = 8). ** p < 0.01 (versus control); ## p < 0.01 (versus model).
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DQJD reduces CCH‐induced neuronal death and inflammation levels in vivo. (A) HE staining of cortex (scale bar = 100 μm) and hippocampal CA1 (scale bar = 100 μm). (B) The number of neurons in the cortex and hippocampal CA1 was evaluated by Image J software ( n = 3). (C) Serum levels of TNF‐α, COX2, and ICAM‐1 were measured by ELISA (mean ± SD, n = 8). ** p < 0.01 (versus control); ## p < 0.01 (versus model).
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DQJD reduces CCH‐induced neuronal death and inflammation levels in vivo. (A) HE staining of cortex (scale bar = 100 μm) and hippocampal CA1 (scale bar = 100 μm). (B) The number of neurons in the cortex and hippocampal CA1 was evaluated by Image J software ( n = 3). (C) Serum levels of TNF‐α, COX2, and ICAM‐1 were measured by ELISA (mean ± SD, n = 8). ** p < 0.01 (versus control); ## p < 0.01 (versus model).
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EIAab Inc mouse cytochrome c oxidase subunit 4 isomers 1, mitochondrial elisa kit (cox iv-1)
DQJD reduces CCH‐induced neuronal death and inflammation levels in vivo. (A) HE staining of cortex (scale bar = 100 μm) and hippocampal CA1 (scale bar = 100 μm). (B) The number of neurons in the cortex and hippocampal CA1 was evaluated by Image J software ( n = 3). (C) Serum levels of TNF‐α, COX2, and ICAM‐1 were measured by ELISA (mean ± SD, n = 8). ** p < 0.01 (versus control); ## p < 0.01 (versus model).
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Image Search Results


DQJD reduces CCH‐induced neuronal death and inflammation levels in vivo. (A) HE staining of cortex (scale bar = 100 μm) and hippocampal CA1 (scale bar = 100 μm). (B) The number of neurons in the cortex and hippocampal CA1 was evaluated by Image J software ( n = 3). (C) Serum levels of TNF‐α, COX2, and ICAM‐1 were measured by ELISA (mean ± SD, n = 8). ** p < 0.01 (versus control); ## p < 0.01 (versus model).

Journal: Journal of Cellular and Molecular Medicine

Article Title: Daqinjiao Decoction Ameliorates CSVD via RXR‐γ/PPAR‐γ/VEGF‐α Pathway: Insights From Transcriptome Sequencing and Network Pharmacology

doi: 10.1111/jcmm.70712

Figure Lengend Snippet: DQJD reduces CCH‐induced neuronal death and inflammation levels in vivo. (A) HE staining of cortex (scale bar = 100 μm) and hippocampal CA1 (scale bar = 100 μm). (B) The number of neurons in the cortex and hippocampal CA1 was evaluated by Image J software ( n = 3). (C) Serum levels of TNF‐α, COX2, and ICAM‐1 were measured by ELISA (mean ± SD, n = 8). ** p < 0.01 (versus control); ## p < 0.01 (versus model).

Article Snippet: Rat ELISA kits of TNF‐α (Lot.EK382/3), COX‐2 (Lot.CSB‐E13399r) and ICAM‐1 (Lot.PI495) were purchased from Multi Sciences (Lianke) Biotech Co. Ltd. (Zhejiang, China), CUSABIO Co. Ltd. (Wuhan, China), and Beyotime Biotech Inc. (Shanghai, China), respectively.

Techniques: In Vivo, Staining, Software, Enzyme-linked Immunosorbent Assay, Control